| 1. | Here , alveolar walls are no longer visible because there is early abscess formation
图中不见肺泡壁，因为早期有脓肿形成。 |
| 2. | The surrounding alveolar walls have capillaries that are dilated and filled with rbc ' s
周围肺泡壁毛细血管扩张充血，血管内充满红细胞。 |
| 3. | Note also that the capillaries in the alveolar walls are congested with many red blood cells
同时也要注意到肺泡壁毛细血管充血，血管内有许多红细胞。 |
| 4. | It can be caused by irreversible expansion of the alveoli or by the destruction of alveolar walls
它是由肺小泡的不可逆的扩张或肺小泡壁被破坏而引起的 |
| 5. | At higher magnification , early abscessing pneumonia is shown . alveolar walls are not clearly seen , only sheets of neutrophils
高倍镜下，可看到早期的脓肿肺炎。肺泡壁不很清楚,仅有嗜中性粒细胞。 |
| 6. | This is normal lung microscopically . the alveolar walls are thin and delicate . the alveoli are well - aerated and contain only an occasional pulmonary macrophage ( type ii pneumonocyte )
正常肺显微镜显示肺泡壁比较薄弱。肺泡内充满空气，有肺巨噬细胞（ ii型上皮细胞） 。 |
| 7. | Conclusions electron microscopic obser vations show that the alveolar epithelial cells , endothelial cells and basement membrane are all injured , interstitial macrophages hyperpplasia , mast cells infiltration and degranulation , electron - dense deposits at alveolar wall are present
结论电镜观察可见特发性肺纤维化时肺泡上皮细胞、血管内皮细胞及基底膜均有损伤，并可见间质巨噬细胞增生，肥大细胞浸润及脱颗粒，肺泡壁电子致密物沉积。 |
| 8. | Results the major morphologic changes were as follows : histologically , alveolar inflammation and interstitial fibrosis were observed . electron microscopic findings were : 1 . alveolar type i cells were degenerated 、 broken - down and desquamated , endothelial cells were swelled , with inter cellular tight junction shortened , alveolar type ii cells hyperplastic , basement membrane thinned and deformed ; 2 . alveolar macrophages and interstitial macrophages were hyperplastic ; 3 . mast cells were infiltrated and degranulated ; 4 . electron - dense deposits were present at alveolar wall ; 5 . myofibroblasts 、 fibroblasts 、 collagen and basement membrane like material were hyperplastic
电镜观察可见： ( 1 ) i型肺泡上皮细胞变性、崩解和脱落，内皮细胞肿胀，细胞间紧密连接短小， ii型肺泡上皮细胞增生，基底膜变薄和破坏； ( 2 )肺泡巨噬细胞、间质巨噬细胞增多； ( 3 )肥大细胞浸润并见脱颗粒现象； ( 4 )肺泡壁电子致密物沉积； ( 5 )肌纤维母细胞、纤维母细胞、胶原原纤维及基底膜样物质增生。 |
